c2 basic confocal microscope Search Results


c2 confocal tokyo  (Nikon)
99
Nikon c2 confocal tokyo
C2 Confocal Tokyo, supplied by Nikon, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti β actin clone c 2 abs  (Santa Cruz Biotechnology)
96
Santa Cruz Biotechnology anti β actin clone c 2 abs
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Anti β Actin Clone C 2 Abs, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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fluorescence confocal microscope nikon c2  (Nikon)
90
Nikon fluorescence confocal microscope nikon c2
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Fluorescence Confocal Microscope Nikon C2, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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confocal microscope nikon az-c2  (Nikon)
90
Nikon confocal microscope nikon az-c2
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Confocal Microscope Nikon Az C2, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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c2 confocal microscope  (Nikon)
90
Nikon c2 confocal microscope
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
C2 Confocal Microscope, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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confocal laser scanning microscope c2  (Nikon)
90
Nikon confocal laser scanning microscope c2
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Confocal Laser Scanning Microscope C2, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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lsm 880 confocal microscope  (Carl Zeiss)
90
Carl Zeiss lsm 880 confocal microscope
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Lsm 880 Confocal Microscope, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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confocal microscopy nikon c2  (Nikon)
90
Nikon confocal microscopy nikon c2
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Confocal Microscopy Nikon C2, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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laser scanning confocal microscope nikon c2-er  (Nikon)
90
Nikon laser scanning confocal microscope nikon c2-er
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
Laser Scanning Confocal Microscope Nikon C2 Er, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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c2 system  (Nikon)
90
Nikon c2 system
Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with <t>anti-β-actin</t> antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).
C2 System, supplied by Nikon, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with anti-β-actin antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).

Journal: Oncotarget

Article Title: Discoidin domain receptor 1 promotes Th17 cell migration by activating the RhoA/ROCK/MAPK/ERK signaling pathway.

doi: 10.18632/oncotarget.10455

Figure Lengend Snippet: Figure 2: DDR1 promotes human Th17 motility in 3D collagen. A. DDR1 expression is reduced by specific DDR1 siRNA (HSS1878780). Polarized Th17 cells were transfected with control or with DDR1 siRNAs and DDR1 expression was assessed by western blot using the anti-DDR1 antibody (C-20). The blot was stripped and reprobed with anti-β-actin antibody to ensure equal loading (top panel). After transfection, the cells were activated with PMA+ionomycin in the presence of brefeldin A to identify IL-17-producing cells, stained with anti-DDR1 and anti-IL-17 antibodies and analyzed by flow cytometry (lower panel). Immunoblot and FACS plots are representative of five independent experiments performed with polarized Th17 cells derived from five different blood donors. B. DDR1 siRNA inhibits Th17 motility in 3D collagen. After transfection, the cells were labelled with calcein AM and embedded in collagen gels. Cell migration was evaluated by live cell confocal microscopy and quantified by computer-assisted cell tracking as described in the “Materials and Methods” section. Representative cell migration tracks over 30 min are presented as x-y projections (distance, in μm) (left panel). The histogram (right panel) represents the mean velocity of 100 cells presented as μm/min. C. DDR1:Fc inhibits Th17 motility in 3D collagen. The cells were embedded in collagen gels containing control human IgG (Fc fragment) or human DDR1:Fc recombinant proteins and cell motility was determined as above. Results (B and C right panels) are mean values ± SD of five independent experiments performed with polarized Th17 cells derived from five different blood donors. *p < 0.05. D. DDR1:Fc inhibits the migratory shape of polarized Th17 cells. Representative photography images from five different experiments of polarized Th17 cells migrating in collagen gels containing either control IgG or DDR1:Fc (400X magnification).

Article Snippet: Non-conjugated rabbit anti-mouse and human DDR1 (clone C-20), non-conjugated rabbit anti-human DDR2 (clone H108) antibodies and the anti-phospho-ERK1/2 (clone E-4), anti-ERK2 (clone C-14), and anti-β-actin (clone C-2) Abs were from Santa Cruz Biotechnology (Santa Cruz, CA).

Techniques: Expressing, Transfection, Control, Western Blot, Staining, Flow Cytometry, Derivative Assay, Migration, Confocal Microscopy, Cell Tracking Assay, Recombinant